Page 103 - WSAVA2018
P. 103

if the content is scheduled to go out well in advance, be sure to monitor activity and engage with posts when necessary. Scheduling posts helps you to bring consistency to each campaign.
It’s simple. Social Media is the way of the future, and the more you put into it, the more you’ll get out of it. By using a calendar, you give your team an opportunity to get
on the same page, and get ahead of the game when it comes to engaging your audience. A veterinary practice is only as strong as the staff and veterinarians behind the scenes. Use all of the tools at your disposal and you’ll start to notice the difference.
Your audience is just waiting to engage with your veterinary practice. The only question left is, what will you share first?
WSV18-0327
SVA INFECTIOUS DISEASES
MANAGEMENT OF PARVOVIRUS INFECTIONS IN DOGS AND CATS
M. Lappin1
1Center for Companion Animal Studies,
Colorado State University, Fort Collins Colorado, USA
Objectives. The primary objectives of this session are to learn updates on the diagnosis, treatment, and prevention of parvovirus infections in dogs and cats.
Canine and feline parvoviruses are non-enveloped DNA viruses which require rapidly divided cells to reproduce. Infections in dogs came from feline panleukopenia
virus and emerged in the late 1970s. Currently, most worldwide dog cases with clinical diseases are infected with CPV-2b or CPV-2c. The small animal parvoviruses are quite resistant to environmental destruction but
are susceptible to bleach. The primary means of transmission is horizontal transmission via oronasal – fecal transmission. Vertical transmission via in utero infection can occur and can leads to myocarditits. CPV- 2b and CPV2c can also infect cats.
CPV-2 first enters the oronasal cavity and infects lymphoid tissue followed by viremia for at least 1-5
days. Rapidly dividing cells of the gastrointestinal tract, myocardium, CNS, skin, kidney and other organs are targeted. Most notably, CPV-2 infects the crypt epithelial cells causing villus blunting. Decreased absorption (manifested as diarrhea), necrosis (sloughing of blood) and inflammation result. Lack of gastrointestinal integrity allows normal GI flora to penetrate into the blood stream and can lead to bacteremia with or without sepsis. Canine parvoviruses are shed primarily in feces for 3
to 14 days post infection, often starting before clinical signs appear. Clinical signs usually develop starting 5 to 12 days after exposure. Dogs with maternal or vaccinal antibodies can usually limit viremia and fully immunized dogs have sterilizing immunity.
Any dog or cat can be infected, but disease is thought
to be more severe in some breeds like the American
Pit Bull Terrier and Rottweilers. Severity of disease depends on virulence of the strain, size of inoculum,
age, breed, and host’s defenses. Clinical signs of CPV
or FPV infection are most severe in puppies or kittens less than 12 weeks that do not have prior immunity. Most dogs have enteritis characterized by foul smelling bloody diarrhea and vomiting. Leukopenia and fever are also common. Cats may have vomiting without diarrhea and sudden death. Both dogs and cats may also have signs of sepsis like red mucous membranes and can develop disseminated intravascular coagulation. CPV-2 can infect the primary CNS with resultant hemorrhage into brain
or spinal cord. In utero infection or infection in pups
Your Singapore, the Tropical Garden City
          101
            









































































   101   102   103   104   105